Originally Posted by
BigTime1
Yes what the state of Iowa would like you tp believe...Truth is that only 2 out of all those animals were CWD positive in both brain and nodes.For a deer to be 100% positive it has to be found in the brain. They only found it in the nodes in the rest which means they only had the gene to carry CWD....Just as people carry the cancer gene without having cancer. That case was a joke in Iowa which is why at court they lost the battle and it cost them a cool 1.6 million to the farmer.
The best part was hearing was hearing them admit that CWD was not a highly contagious disease. Under oath in front of the judge. That farm is no longer under lock down and they are free to use their property again! As they should. States cant just yank peoples land away from them and say they are locked from using it as they see fit. Hence the 1.6 of taxpayers monies lost by that states strong arming of private citizens. Cost them a pile!
Man you are 100% laughable. Just where in the world do you get your misinformation from? There has NOT been a genetic barrier nor genetic susceptibility found in whitetail deer. The ONLY related information is here in this research which shows that there are SOME variations that will SLOW the PROGRESSION of the disease but NOTHING has yet to be identified that will STOP it on a genetic level. This is research from the Journal of General Virology and the study was conducted through Wisconsin's Dept of animal health and Biomedical sciences, at University of Wisconsin and the Veterinary Diagnostic Lab in Wisconsin.
The primary sequence of the prion protein affects susceptibility to transmissible spongiform
encephalopathies, or prion diseases, in mice, sheep and humans. The Prnp gene sequence of
free-ranging, Wisconsin white-tailed deer was determined and the Prnp genotypes of chronic
wasting disease (CWD)-positive and CWD-negative deer were compared. Six amino acid changes
were identified, two of which were located in pseudogenes. Two alleles, a QRK polymorphism
at codon 226 and a single octapeptide repeat insertion into the pseudogene, have not been
reported previously. The predominant alleles – wild-type (Q95, G96 and Q226) and a G96S
polymorphism – comprised almost 98 % of the Prnp alleles in the Wisconsin white-tailed deer
population. Comparison of the allelic frequencies in the CWD-positive and CWD-negative deer
suggested that G96S and a Q95H polymorphism were linked to a reduced susceptibility to CWD.
The G96S allele did not, however, provide complete resistance, as a CWD-positive G96S/G96S
deer was identified. The G96S allele was also linked to slower progression of the disease in
CWD-positive deer based on the deposition of PrPCWD in the obex region of the medulla oblongata.
Although the reduced susceptibility of deer with at least one copy of the Q95H or G96S allele
is insufficient to serve as a genetic barrier, the presence of these alleles may modulate the impact of
CWD on white-tailed deer populations.
Now, you can fall back on your little man in a lab coat running around infecting deer again since yet ANOTHER one of your bogus claims is shot down. Keep trying kiddo. You might stumble into the light of day sometime.